- Genetic evaluation of your Alzheimer's risk
- Analysis of the APOE types for risk assessment
- Personalized prevention program, which can delay the development of the disease for years
- Adapted nutrition to reduce the risk
- Micronutrient recommendation for reducing the risk
- Analysis of over 18 genetic variations predicting the effectiveness of over 20 drugs
- More effective treatment through the optimal use of medication
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Alzheimer's disease is a disease characterized by a progressive loss of certain brain cells. The cause of Alzheimer's is not fully understood. However, certain genetic traits have been clearly linked to a significantly increased risk of developing the disease. These traits cause abnormally folded proteins to accumulate in certain regions of the brain and allow for the development of large numbers of toxic molecules known as free radicals that damage brain cells. The damaged brain cells in affected regions of the brain slowly deteriorate.
Early signs of Alzheimer's can be detected as early as eight years before diagnosis. Examples of early symptoms include short term memory loss and difficulties with language as well as depression and apathy. The disease is often not recognized until the person develops noticeable learning disorders and short term memory loss increases while long term memory remains unaffected. In advanced stages, persons diagnosed with Alzheimer's completely lose even basic skills and abilities and eventually cease to recognize close friends and family or even day-to-day objects. Irritability and aggression are common and as the disease progresses, the person becomes increasingly dependent on caregivers.
Alzheimer's disease accounts for roughly 60 percent of the roughly 24 million diagnosed cases of dementia worldwide. The most common form affects individuals over the age of 65. Around 2% of 65-year-olds are affected whereas among 70-year-olds the figure rises to 3%. 6% of 75-year-olds and roughly 20% of 85-year-olds display symptoms of the disease.
So far, the scientific community has not found a cure for Alzheimer's disease. However, there are a great number of preventative measures that can be effective for people with a genetic predisposition to Alzheimer's. Memory training, changes in lifestyle, an appropriate diet and controlling certain other conditions can all play an important role in preventing Alzheimer's. Measures such as these can delay the development of Alzheimer's for many years or even prevent it entirely. It is therefore especially important for persons who carry these genetic defects to learn about their risk and begin preventative measures as early as possible.